
May 28th 05, 05:24 PM
|
|
Once in a while you get these little gems that are worth filing in the ol'
grey matter. The rest of the time, I run the usenet shredder almost
continuously.
"Dave S" wrote in message
k.net...
wrote:
Hey dave,
Ever swam under water?
Ever hypervent your self before taking that last breath so you can stay
down longer?
What would you call that?
You are eliminating the Carbon Dioxide in your body to VERY low levels,
which helps forestall the URGE to breath until they build back up to
higher levels.
Hyperventilation does not increase oxygen capacity over and above what
normal breathing does. It simply clears out waste products. All of this is
again a function of the partial pressures and pressure gradients that were
discussed elsewhere in this thread.
On room air, your pO2 in arterial blood is in the area of 80-100 mmhg.
Your pC02 is in the neighborhood of 40 mmhg. Oxygen Saturation is in the
90-100% range.
Mixed venous blood gases (which are sampled from the pulmonary artery, in
patients with the proper monitoring equipment) on room air tends to be in
the 40-50 mmHg range. This corresponds with about ONE of the FOUR oxygen
molecules on each hemoglobin molecule being extracted, and a saturation in
the 60-75% range. Without a fresh supply of oxygen in the lungs to extract
from, this "desaturated" blood makes a second trip around, and because of
the laws of nature, it is even more difficult for the remaining oxygen to
be extracted. For practical purposes, its essentially NOT going to release
more than two of the four hemoglobin-bound oxygen molecules. Your arterial
oxygen saturation gets too low and you will end up getting confused,
euphoric, or just unconscious.
Hyperventilation in an otherwise healthy individual will not increase the
p02, but can/will drive the pCO2 in arterial blood down to the 20's.
Prolonged hyperventilation will cause vasoconstriction in the brain (as
well as everywhere else), resulting in lightheadedness, dizziness,
cramping in the fingers/toes, and chest pain. I deal with persons on
breathing machines EVERY DAY for a living. If their oxygen level is low,
we dont turn up the rate or volume, we turn up the oxygen concentration.
If their CO2 level is high, THEN we turn up rate/volume to increase the
amount of VENTILATION. Ventilation is the removal of waste products.
Oxygenation is the delivery of oxygen. They are TWO completely different
functions, and not very related to each other, despite the fact they are
happening in the same place at the same time.
It is still possible to pass out from Hypoxia without building up CO2
levels back to the amount needed to trigger the chemoreceptors to tell
your brain "I GOTTA BREATHE". It has been the cause of more than one
drowning/diving accident. If you want further reference on this, a
knowledgeable professional diver, an aerospace medicine MD or a hyperbaric
technician should be able to validate this information.
Dave
|